Such patients can live only in

Such patients can live only in the event that adequate shunting through defect of an interatrial septum (the best variant), defect of an interventricular septum or a persistent arterial duct takes place. If ventricles also are located (so-called a transposition cheap ultracet of the main arteries) the life is possible and without blood shunting since the morphological right ventricle refers blood to an aorta, and the morphological left ventricle throws out venous blood in a pulmonary artery. 8. At the venous ventricle (under the morphological characteristic corresponding to a left ventricle [LV]) refers a transposition of the main arteries blood to a pulmonary artery [], and the arterial ventricle (having an anatomic structure of a right ventricle [RV]) throws out blood in an aorta [] and is compelled to develop adequate pressure. We will notice, that the diverticulum of the left upper edge of a shade of heart on the roentgenogram of organs of a thorax easily can be wrongly accepted for a contour of a pulmonary artery Century At the second warm tone usually is not split and formed by exclusively aortal component of the second warm tone. The reasons leading to weakening of a pulmonary component, are: 1.
Deformation of the valve of a pulmonary artery in the presence of a valval stenosis (meets approximately in one third of cases ) 2. A forward locating of the enlarged aorta in relation to the narrowed pulmonary artery laying more dorsally 3. Low pressure in a pulmonary artery which is caused by abnormal dump of blood through high subaortal defect of an interventricular septum immediately in an aorta. The reasons of the raised loudness of aortal or pulmonary components of the second warm tone 1. What physiological and pathological conditions can lead to what the aortal component of the second tone becomes louder, than in norm? And. Diseases at which systolic arterial pressure (for example, at a system arterial hypertonia) raises. Last is sometimes accompanied by occurrence "drum-type" () the second cardiac sound. The note: The tambourine (tambourine) is the small drum closed only on the one hand. "Drum-type" second warm tone is often auscultated at an arterial hypertonia. It also has been described at some colchicine side effects patients with expansion of proximal department of the aorta, leading to a tension of cusps of the aortal valve. At patients with an arterial hypertonia "drum-type" second tone can disappear at fast short depression of arterial pressure (for example, after amyle nitrite reception), but can remain despite proof depression of arterial pressure against hypotensive therapy. . Conditions for which the hyperkinetic type of a peripheric circulation (for example, young age, a thyrotoxicosis and an aortal regurgitation) is characteristic. The note: Loudness of a closing snap of the aortal valve is defined by a return stream of blood in an aorta and rate of reduction of a left ventricle during a systole. Differently, loudness of an aortal component depends on contractility of a left ventricle and, hence, can be normal at persons with an arterial hypertonia and a circulatory unefficiency. Century Any pathological conditions at which the aorta settles down near to a forward thoracal wall, including a dilatation of proximal department of an aorta, its locating in front from a pulmonary artery (as at a transposition of the main vessels) or behind a pulmonary trunk. 2. What physiological and pathological conditions, besides a thin thoracal wall and a pulmonary hypertensia, can lead to what the pulmonary component of the second tone becomes louder, than in norm? Diseases at which the blood flow augmentation in a pulmonary artery (for example, defect of an interatrial or interventricular septum) is observed. Notes: And. The raised pulmonary blood flow at defect of an interatrial septum not always leads to that the pulmonary component of the second warm tone becomes louder than the aortal. The exception is made only by those cases when pressure in a pulmonary artery reaches 50 mm hg and more. If defect of an interatrial septum is combined with a stenosis of a pulmonary artery in most cases the pulmonary component will be more silent aortal, and the interval between them will be enlarged and will make 60 or more. . Absolute loudness of a pulmonary component is a good sign of a pulmonary hypertensia at a mitral stenosis only in the event that it is great. On the other hand, louder (in comparison with aortal) the pulmonary component in the second at the left also is a good sign of a pulmonary hypertensia, however in any way does not reflect degree of expression of last.