PERIPHERIC

PERIPHERIC (FLACCID, ATROPHIC) THE PARALYSIS

The flaccid paralysis is, as soon as that has been told, result of a lesion of peripheric impellent neurones, i.e. Cells of forward horns of a spinal cord (or impellent kernels order ultram 400mg of cranial nerves), lobbies. Roots and impellent fibers of spinal and cranial peripheric nerves. This type of paralyses is characterised by loss of reflexes, a hypotension and the degenerate atrophy of muscles accompanied by a so-called reaction of degeneration.
Loss of reflexes (or their weakening at an incomplete lesion) becomes clear if we recollect, that the peripheric impellent neurone is at the same time and a centrifugal, efferent part of a reflex arch. At a break of any department of last the reflex certificate is impossible or (at an incomplete break) is weakened.
The atony or hypotension of muscles speaks also a break of a reflex arch therefore the muscle loses inherent to it constant, so-called a tonus supported in norm by the same reflex arch. Besides, the atony can be strengthened an arising atrophy of muscular mass. Atonic muscles are to the touch flabby, flaccid, passive movements are superfluous, joints "are stirred up". Such condition of a musculation gives the basis to name a flaccid paralysis also flaccid, or atonic.
The atrophy of muscles results from dissociation with a cell of a forward horn, whence on an impellent nervous fiber to a muscle притекают the nervously-trophic impulses stimulating a normal exchange of a muscular tissue. Presence of muscular atrophies causes one more definition of a flaccid paralysis - as atrophic.
The atrophy of muscles comes after a degeneration and destruction of nervous impellent fibers, there is muscle "denervation". As a result in nerves impellent fibers disappear from top to bottom from a break place; in a muscle the degenerate process characterised by changes of muscular fibers, their destruction, development of a fatty and connecting tissue develops.
There are characteristic changes of electric reactions of the amazed nerves typical for a flaccid paralysis and the muscles, named a reaction of degeneration or degenerations ().
In norm at a nerve boring galvanic (at short circuit and disconnection) and faradic reduction иннервируемых it of muscles occurs currents; at a boring its reduction occurs the same currents of immediately muscle also, and on a galvanic current it arises extremely quickly ("immediately") and differs that катодозамыкательное reduction more than анодозамыкательное (the gas station).
At a reaction of degeneration (degeneration) the nerve does not spend a current to a muscle for its impellent centrifugal fibers перерождены and were lost; the muscle is denervated and loses ability of reduction on a boring a faradic current, keeping excitability only on the galvanic. But also this reduction becomes slow («червеобразным»), and big becomes already vpxl 400mg reduction (the gas station>). Such condition is called as a full reaction of degeneration and attacks 12 - 15th day after a break of a nerve or destruction of a cell of a forward horn.
At an incomplete lesion of a peripheric impellent neurone there can come a partial reaction of degeneration when excitability of a nerve on both currents is not lost, and is only weakened, no less than faradic excitability of a muscle; muscle reduction at a boring a galvanic current also arises slowly, with prevalence анодозамыкательного effect over катодозамыкательным (the gas station>).
The full reaction of degeneration yet is not a bad prognostic sign: under condition of restoration (neogenesis) of a nervous fiber it can be replaced through a phase of partial reaction with normal electroexcitability. But if the muscle at a flaccid paralysis remains completely denervated from above 12 - 14 months (sometimes and more longly) as a result of a progressing degeneration of muscular fibers they perish completely, are replaced with a fatty and connecting tissue, and there comes a cirrhosis of a muscle with loss already and its reactions to a galvanic current, i.e. Full loss of electroexcitability develops. Last specifies in irreversibility происшедших in a muscle of changes.